Dihydromyricetin is mostly extracted from a woody vine of the genus Agave in the grape family, and is also extracted from the jujube. The main active ingredient is flavonoids, which have many unique effects such as scavenging free radicals, anti-oxidation, anti-thrombosis, anti-tumor, and anti-inflammatory. Dihydromyricetin is a relatively special flavonoid. In addition to the general characteristics of flavonoids, it also has the effects of relieving alcohol poisoning, preventing alcoholic liver and fatty liver, inhibiting liver cell deterioration, and reducing the incidence of liver cancer. It is a good product for protecting the liver and sobering up.
The Function of Dihydromyricetin Dihydromyricetin is an extract of the grape plant rattan tea, and is the main active ingredient of rattan tea, a flavonoid compound. This type of substance has many unique effects such as scavenging free radicals, anti-oxidation, anti-thrombosis, anti-tumor, and anti-inflammatory. Dihydromyricetin is a relatively special flavonoid compound, which has special effects in relieving alcohol poisoning, preventing alcoholic liver and fatty liver, inhibiting liver cell deterioration, reducing the incidence of liver cancer, anti-hypertension, inhibiting platelet aggregation in vitro and thrombosis in vivo, lowering blood lipid and blood sugar levels, improving SOD activity, and protecting the liver.
Antibacterial effect: Pharmacological experiments show that dihydromyricetin has an antibacterial effect on Bacillus subtilis, Staphylococcus aureus, Salmonella, Escherichia coli, Bacillus aerogenes, Saccharomyces cerevisiae, Rhodotorula glutinosus, Penicillium, Aspergillus niger, Aspergillus flavus, Mucor and Rhizopus, especially against Gram-positive and Gram-negative cocci or bacilli.
Regulation of blood sugar and blood lipids: Pharmacological experiments on mice gavage showed that dihydromyricetin can significantly inhibit the increase of blood sugar in mice induced by alloxan, adrenaline, streptozotocin, etc., and at the same time increase serum insulin levels, significantly reduce lymphocyte infiltration in pancreatic tissue, significantly reduce inflammatory response, and significantly increase the number of pancreatic islets. Dihydromyricetin has a lowering effect on the serum triglyceride (TG) content of streptozotocin-induced hyperglycemic rats, and has no significant effect on serum total cholesterol (TC) and high-density lipoprotein cholesterol (HDL2C) content.
Liver protection effect: Dihydromyricetin has a significant protective effect on carbon tetrachloride poisoning damage of rat hepatocytes cultured in vitro, and D2 galactosamine and lipopolysaccharide-induced mouse liver damage. Dihydromyricetin can protect the liver, accelerate the rapid decomposition of acetaldehyde, a product of ethanol metabolism, into non-toxic substances, and reduce its damage to liver cells. In addition, dihydromyricetin can improve the increase in serum lactate dehydrogenase activity caused by liver cell damage, inhibit the formation of hepatic M cell collagen fibers, thereby protecting the liver, greatly reducing the damage of ethanol to the liver, and quickly restoring the liver to normal state. Microherb experiments have shown that dihydromyricetin can protect the liver, accelerate the rapid decomposition of acetaldehyde, a metabolite of ethanol, into non-toxic substances, and reduce damage to liver cells. Dihydromyricetin has a rapid onset and a long-lasting effect. It is a good product for protecting the liver and sobering up.
Antioxidant effect: Dihydromyricetin with a purity of 98% can significantly inhibit the generation of malondialdehyde (MDA) in rat myocardial, liver and brain tissue homogenates, and the effect of inhibiting MDA generation increases with the increase of dihydromyricetin concentration. The scavenging rate of diphenyltrinitrophenylhydrazine (DPPH) free radicals in the test system by dihydromyricetin with a content of 99%. Dihydromyricetin can significantly inhibit the formation of MDA in oils and fats. As the purity of dihydromyricetin increases (60% to 90%), the antioxidant effect increases. It has a strong antioxidant effect on both animal and vegetable oils.
Anti-tumor effect: Anti-tumor pharmacodynamic studies have found that one of its active ingredients, the small molecule compound of ampelopsis, has a relatively strong anti-cancer effect. In vitro anti-tumor effect studies have found that the compound can achieve the purpose of improving tumor treatment effects and improving patient vital signs by inhibiting tumor blood vessel growth, regulating and enhancing cellular immune function, etc., and has shown significant tumor inhibition in tumor model experiments such as leukemia and nasopharyngeal carcinoma.
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